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MIDOSTAURIN PLUS INTENSIVE CHEMOTHERAPY VERSUS INTENSIVE CHEMOTHERAPY IN FLT3 MUTATED ACUTE MYELOID LEUKEMIA. A RWE STUDY
Author(s): ,
Adolfo De La Fuente
Affiliations:
Hematology,MD Anderson CC Madrid,Madrid,Spain
,
David Martinez-Cuadron
Affiliations:
Hematology,H. Universitario y Politecnico La Fe Valencia,Valencia,Spain
,
Cristina Gil
Affiliations:
Hematology,H. General Universitario de Alicante,Alicante,Spain
,
Teresa Bernal
Affiliations:
Hematology,H. U. Central de Asturias,Oviedo,Spain
,
Lorenzo Algarra
Affiliations:
Hematology,H. G. U. de Albacete,Albacete,Spain
,
Carlos Rodriguez-Medina
Affiliations:
Hematology,H. U. de Gran Canaria Doctor Negrin,Las Palmas de Gran Canaria,Spain
,
Jose Antonio Perez-Simon
Affiliations:
Hematology,H. Virgen del Rocio,Sevilla,Spain
,
Mar Tormo
Affiliations:
Hematology,H. Clinico Universitario INCLIVA,Valencia,Spain
,
Susana Vives
Affiliations:
Hematology,ICO H. Germans Trias i Pujol,Badalona,Spain
,
Raimundo Garcia-Boyero
Affiliations:
Hematology,H. G. U. de Castellon,casstellon de la Plana,Spain
,
Josefina Serrano
Affiliations:
Hematology,H. U. Reina Sofia IMIBIC,Cordoba,Spain
,
Mercedes Colorado
Affiliations:
Hematology,H. U. Marques de Valdecilla,Santander,Spain
,
Juan Bergua
Affiliations:
Hematology,H. San Pedro de Alcantara,Caceres,Spain
,
Jorge Labrador
Affiliations:
Hematology,H. U. de Burgos,Burgos,Spain
,
Mariluz Amigo
Affiliations:
Hematology,H. U. Morales Meseguer,Murcia,Spain
,
Pilar Martinez-Sanchez
Affiliations:
Hematology,H. U. 12 de Octubre,Madrid,Spain
,
Juan Ignacio Rodriguez-Gutierrez-Macias
Affiliations:
Hematology,H. U. Basurto,Bilbao,Spain
,
Maria Jose Sayas
Affiliations:
Hematology,H. U. Doctor Peset,Valencia,Spain
,
Aurelio Lopez
Affiliations:
Hematology,H. Arnau de Vilanova,Valencia,Spain
,
Carlos Javier Cerveró
Affiliations:
Hematology,H. Virgen de la Luz,Cuenca,Spain
,
Esperanza Lavilla-Rubira
Affiliations:
Hematology,H. Lucus Augusti,Lugo,Spain
,
Mayte Olave
Affiliations:
Hematology,H. Lozano Blesa,Zaragoza,Spain
,
Fernando Ramos
Affiliations:
Hematology,H. U. de Leon,Leon,Spain
,
Guiomar Bautista
Affiliations:
Hematology,H. U. Puerta de Hierro de Majadaonda,Madrid,Spain
,
Grasa Vasconcelos
Affiliations:
Hematology,H. Santa Maria,Lisboa,Portugal
,
Paola Beneit
Affiliations:
Hematology,H. San Juan de Alicante,Alicante,Spain
,
Antonio Diaz-Lopez
Affiliations:
Hematology,Fundacion MD Anderson,Madrid,Spain
Pau Montesinos
Affiliations:
Hematology,H. Universitario y Politecnico La Fe Valencia,Valencia,Spain
EHA Library. De La Fuente A. 06/09/21; 325201; EP447
Dr. Adolfo De La Fuente
Dr. Adolfo De La Fuente
Contributions
Abstract
Presentation during EHA2021: All e-poster presentations will be made available as of Friday, June 11, 2021 (09:00 CEST) and will be accessible for on-demand viewing until August 15, 2021 on the Virtual Congress platform.

Abstract: EP447

Type: E-Poster Presentation

Session title: Acute myeloid leukemia - Clinical

Background
Introduction. Midostaurin has been approved by EMA and FDA in combination with IC for FLT3-mutated AML based on improved overall survival noted in the RATIFY phase 3 trial (Stone et al, N Engl J Med 2017) The experience with midostaurin in day to day practice is limited.

Aims

Aim: The aim of this study is to analyze effectiveness and tolerability of midostaurin plus intensive chemotherapy (midos+IC) vs intensive chemotherapy alone (IC) as first lines in AML FLT3 positive patients and to identify risk factors.

Methods

Methods: We carried out the analysis on previously untreated AML FLT3 mutated patients included in PETHEMA AML epidemiological registry (NCT02607059). Selection criteria for the study were as follows: Age >18, diagnosis of AML FLT3 positive (ITD and TKD) under WHO criteria, treated with midos+IC or IC (IC defined as 7+3 induction and HiDAC or intermediate dose AraC consolidations) during the period 01/01/2010 to 01/03/2020, patients who died before day 7 of treatment were excluded. Response were assessed using the ELN-2010 criteria, toxicity by the CTCAE v4.0 scale, OS by Kaplan-Meier and early mortality as mortality within the first 8 weeks (M8wks).

Results

Results: A total of 385 of previously untreated AML FLT3 positive patients were analyzed (54 treated with Midos+IC and 331 with IC), median age 52yrs (20-70) and 53yrs (18-70), ECOG ≥2: 18.5% and 12.7% respectively. Baseline characteristics are summarized in table 1. Antifungal triazole prophylaxis was the standard in both cohorts. Early mortality M8wks was 3.7% and 6.6% and complete response rate (CR after 1 or 2 Inductions) 86% vs  66% (p 0.005) for midos+IC and IC respectively.


Overall survival is significantly longer in the mido+IC group than in the IC group (not reached vs 19 months, p=0.022), 24mOS 79.2% vs 54.2% (p0.026).


We observed a trend of benefit in all the following subgroups, a: FLT3 ITD low with NPM1 mut, b: FLT3 ITD low with NPM1 wt plus FLT3 ITD high with NPM1 mut; and c: FLT3 high with NPM1 wt. In our study we observe in midos+IC cohort a higher rate of patients consolidated in CR1 with alloSCT and a significantly longer OS for these patients (42.5% vs 28.7%) and (not reached vs 44.7 months).

Conclusion

Conclusions: The results of this study confirm in a real-world setting that midostaurin associated with IC improve the outcome of AML FLT3 positive patient compared versus IC alone.

Keyword(s): FLT3, Flt3 inhibitor, Leukemia

Presentation during EHA2021: All e-poster presentations will be made available as of Friday, June 11, 2021 (09:00 CEST) and will be accessible for on-demand viewing until August 15, 2021 on the Virtual Congress platform.

Abstract: EP447

Type: E-Poster Presentation

Session title: Acute myeloid leukemia - Clinical

Background
Introduction. Midostaurin has been approved by EMA and FDA in combination with IC for FLT3-mutated AML based on improved overall survival noted in the RATIFY phase 3 trial (Stone et al, N Engl J Med 2017) The experience with midostaurin in day to day practice is limited.

Aims

Aim: The aim of this study is to analyze effectiveness and tolerability of midostaurin plus intensive chemotherapy (midos+IC) vs intensive chemotherapy alone (IC) as first lines in AML FLT3 positive patients and to identify risk factors.

Methods

Methods: We carried out the analysis on previously untreated AML FLT3 mutated patients included in PETHEMA AML epidemiological registry (NCT02607059). Selection criteria for the study were as follows: Age >18, diagnosis of AML FLT3 positive (ITD and TKD) under WHO criteria, treated with midos+IC or IC (IC defined as 7+3 induction and HiDAC or intermediate dose AraC consolidations) during the period 01/01/2010 to 01/03/2020, patients who died before day 7 of treatment were excluded. Response were assessed using the ELN-2010 criteria, toxicity by the CTCAE v4.0 scale, OS by Kaplan-Meier and early mortality as mortality within the first 8 weeks (M8wks).

Results

Results: A total of 385 of previously untreated AML FLT3 positive patients were analyzed (54 treated with Midos+IC and 331 with IC), median age 52yrs (20-70) and 53yrs (18-70), ECOG ≥2: 18.5% and 12.7% respectively. Baseline characteristics are summarized in table 1. Antifungal triazole prophylaxis was the standard in both cohorts. Early mortality M8wks was 3.7% and 6.6% and complete response rate (CR after 1 or 2 Inductions) 86% vs  66% (p 0.005) for midos+IC and IC respectively.


Overall survival is significantly longer in the mido+IC group than in the IC group (not reached vs 19 months, p=0.022), 24mOS 79.2% vs 54.2% (p0.026).


We observed a trend of benefit in all the following subgroups, a: FLT3 ITD low with NPM1 mut, b: FLT3 ITD low with NPM1 wt plus FLT3 ITD high with NPM1 mut; and c: FLT3 high with NPM1 wt. In our study we observe in midos+IC cohort a higher rate of patients consolidated in CR1 with alloSCT and a significantly longer OS for these patients (42.5% vs 28.7%) and (not reached vs 44.7 months).

Conclusion

Conclusions: The results of this study confirm in a real-world setting that midostaurin associated with IC improve the outcome of AML FLT3 positive patient compared versus IC alone.

Keyword(s): FLT3, Flt3 inhibitor, Leukemia

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